I 20.0 – Unstable angina – example of writing a call card

What is angina

Angina pectoris is a specific pain syndrome characteristic of myocardial ischemia. There are stable and unstable angina, as well as exertional and resting angina.

Stable angina is one that occurs in an unchanged form. We can say that this is a “habitual” symptomatology for a person. However, this situation cannot be considered the norm: serious treatment is required, often surgical.

Unstable angina is either angina that has arisen for the first time, or one where the nature of the pain syndrome and sensations differs significantly from the usual state. This is an extremely dangerous condition, fraught with a high risk of developing a heart attack, and requiring an immediate call to the ambulance.

Angina pectoris is one of the manifestations of atherosclerosis and coronary heart disease, which is manifested by severe chest pain during physical activity and stress. However, chest pain is not always a manifestation of angina.

It is important to note symptoms such as:

• squeezing, pressing or burning nature of the attack;
• impact under the left shoulder blade or in the arm, neck, lower jaw;
• lack of oxygen and dizziness;
• obvious interruptions in the functioning of the heart: fluctuations in heart rate and blood pressure.

Most often, pain with angina pectoris manifests itself during walking, climbing stairs, physical effort, and stress. The attack lasts from 1 to 15-20 minutes and is facilitated by taking nitroglycerin. Symptoms may appear all at once or separately.

Angina at rest, unlike the previous type, is characterized by a sudden and prolonged attack, which is activated without external provoking factors, sometimes during sleep. In this case, we can add to the main symptoms: an uncontrollable panic attack, a pronounced feeling of suffocation, a violation of the autonomic functions of the body. We will write more about this type of angina in the following articles.

In addition to extremely unpleasant symptoms, such manifestations indicate a high risk of myocardial infarction, as well as the development of heart failure.

Cardiovascular diseases claim half of human lives - exactly the same as all other causes combined.

I 20.0 – Unstable angina – example of writing a call card

Patient, 64 years old

Reason for calling – Chest pain (history of coronary artery disease)

Complaints, medical history

Complaints of general weakness, pressing pain behind the sternum, burning sensation radiating to the left shoulder, back (between the shoulder blades), sweating, feeling of lack of air.

The feeling worsened for about 6 hours, the pain arose at rest, intensified with the slightest physical activity, short-term relief after taking isoket spray. I took isoket spray about 10 doses, bisoprolol 10 mg, lisinopril 10 mg, cardiomagnyl 1 tablet. This is not the first time this condition has happened; he was previously treated as an inpatient. During physical activity, she notes infrequent short-term attacks of chest pain, which are relieved with rest after taking nitrates, after 5-10 minutes.

Anamnesis of life

Hypertension stage III. IBS. PEAKS (2012). Diabetes mellitus type II. Cerebrovascular disease.

Regularly takes enalapril 10 mg/day, bisoprolol 10 mg/day, cardiomagnyl 1 tablet/day, maninil 1 tablet/day.

Comfortable blood pressure – 140/90

Physical examination

Condition: moderate;

Consciousness – clear, Glasgow scale – 15 points, behavior – calm;

Pupils – normal, D = S, reaction to light – lively, no gaze paresis, no nystagmus;

Skin – pale, dry, clean;

Heart sounds are muffled, rhythmic, no murmurs. The pulse in the peripheral arteries is of satisfactory quality, rhythmic;

Nervous system – without pathology, no meningeal symptoms;

The pharynx is calm, the tonsils are normal;

Excursion of the chest - normal, type of breathing - normal, percussion - pulmonary sound, auscultation - vesicular breathing, no shortness of breath;

There is no peripheral edema;

The tongue is clean and moist. The abdomen is soft, painless, participates in the act of breathing, there are no symptoms of peritoneal irritation, the liver is not enlarged, stool is formed, 1 time per day;

Urine output is normal, SSPO is negative.

Main pathology

Consciousness is preserved, the patient is adequate. The skin is pale and dry. Heart sounds are muffled and rhythmic. The pulse in the peripheral arteries is of satisfactory quality, rhythmic.

Blood glucose – 6.5 mmol/l

Troponin test – negative

Electrocardiography

15-35 – Sinus rhythm 86 per minute. Signs of left ventricular hypertrophy. Signs of subepicardial damage to the anterior wall (ST depression I, II, aVL, V1-V4 1-2 mm). Negative dynamics with previous ECGs.

15-50 – Sinus rhythm 64 per minute. No dynamics from the previous ECG.

16-30 – Sinus rhythm 64 per minute. No dynamics from the previous ECG.

Diagnosis by the EMS team

IBS. Unstable angina III B (Braunwald). Post-infarction cardiosclerosis.

Therapeutic measures

• oxygen inhalation through nasal catheters
• 15-36 – peripheral vein catheterization
• 15-37 – Isoket spray s/l 1 dose every 5 minutes – 4 doses (no effect)
• 15-38 – Tav. Acidi acetylsalicylici 250 mg p/os
• 15-38 – Tav. Plavix 300 mg p/os
• 15-39 – Sol. Clexani 4000 ED IV
• 15-42 – Sol. Betaloc 10 mg IV
• 15-45 – Sol. Morphini hydrochloride 1% – 1 ml intravenously in fractions
• ECG monitoring
• hospitalization on a stretcher to the regional vascular center.

Treatment result

Improvement. The pain syndrome was relieved by the administration of morphine and did not recur during transportation.

Causes of angina

The cause of angina is the partial blocking of the lumen of the arteries by atherosclerotic plaques, resulting in severe oxygen starvation of the heart cells, which causes pain. An additional role is played by vasospasm, which can be provoked, for example, by nervous strain or sudden exposure to cold.

It is the presence of atherosclerotic plaques that is the leading factor in the development of angina pectoris. The development of coronary heart disease is a complex and multicomponent process, which is provoked and accelerated due to risk factors:

• arterial hypertension,
• smoking, obesity,
• cholesterol metabolism disorder,
• diabetes,
• low physical activity and poor nutrition.

In the case of atherosclerosis, angina begins to manifest itself when an atherosclerotic plaque fills the artery by more than 50% - preventing the normal passage of blood through the heart and, accordingly, the saturation of the heart muscle with oxygen. This leads to the development of an imbalance in the functioning of the heart.

With physical and psycho-emotional stress, the situation worsens. The heart begins to work more actively - it contracts more often and stronger. In this state, it requires more oxygen and nutrients. However, due to vasoconstriction, this need is not satisfied, which leads to oxygen starvation and, as a result, pain.

We feel pain thanks to the instinct of self-preservation. She is a kind of alarm signal. Therefore, any pain attack is a rational reason to consult a doctor.

Unstable angina

Unstable angina is a concept under which several clinical situations are united: their common feature is the presence of attacks of pain in the chest.

Recently, unstable angina has been defined as an exacerbation of coronary heart disease (impaired blood circulation in the arteries designed to provide the heart muscle (myocardium) with the necessary supply of blood), threatening the development of myocardial infarction (death of a section of the heart muscle due to impaired circulation in it) or sudden death.

Symptoms of unstable angina

The main symptom of unstable angina is pain:

• the nature of the pain is quite strong, squeezing or pressing, often a feeling of heaviness or lack of air is felt;
• localization (location) of pain - behind the sternum or in the precordial region, that is, along the left edge of the sternum; pain radiates to the left arm, left shoulder or both arms, neck area, lower jaw, between the shoulder blades, left subscapular area;
• duration – more than 10 minutes.

Factors causing pain:

• it is impossible to determine a clear connection between pain and certain physical activity or other conditions (for example, pain appears in windy weather or after eating), a painful attack often occurs at rest or with minimal physical activity (bending over, walking around the apartment);
• the end of a painful attack - often after repeated administration of nitroglycerin;
• attacks occur more frequently than before.

Heart rhythm disturbances gradually develop:

• increased heart rate, palpitations;
• heartbeats become irregular and intermittent.

Shortness of breath occurs, which occurs with minimal physical activity and even at rest, and a feeling of lack of air.

Forms

There are several forms of unstable angina:

• new-onset angina (a disease manifested by discomfort or pain in the chest of a compressive, pressing nature, which is most often localized behind the sternum and can radiate (give) to the left arm, neck, lower jaw, epigastric (“under the stomach”) region);
• progressive angina (increasing intensity and/or duration of painful attacks, their occurrence with significantly less physical activity or at rest, the addition of shortness of breath, feeling of lack of air);
• post-infarction angina (occurring 24 hours and up to 8 weeks after the development of myocardial infarction (death of part of the heart muscle due to cessation of blood flow in this area)) or postoperative angina (within one to two months after a successful operation to restore blood flow).

​In practice, the Braunwald classification (1989) is often used, dividing unstable angina into three classes (the higher the functional class, the greater the likelihood of developing complications:

• Class I - new angina or worsening of existing angina within a month;
• Class II - angina at rest during the previous month;
• Class III - angina at rest during the last 48 hours.

Depending on the conditions of occurrence, the following are also distinguished:

• class A - secondary unstable angina. This group includes patients in whom unstable angina develops in the presence of factors that aggravate ischemia (anemia (anemia), increased body temperature, infection, low blood pressure, uncontrolled hypertension, heart rhythm disturbances, emotional stress and others);
• class B - primary unstable angina. This group includes patients in whom unstable angina develops without extra-cardiac conditions that increase ischemia;
• class C - post-infarction unstable angina. This group includes patients in whom angina develops in the first 2 weeks of myocardial infarction.

Causes

The main cause of unstable angina is the rupture of the so-called unstable atherosclerotic plaque (a formation consisting of a mixture of fats (primarily cholesterol (a fat-like substance that is a “building material” for the body’s cells) and calcium), which provokes the formation of a blood clot with incomplete closure of the lumen of the vessel .

The presence of a thrombus (blood clot) in the artery supplying the heart prevents adequate blood supply to the heart muscle, which leads to the appearance of pain and a full-blown clinical picture (set of symptoms) of unstable angina.

Plaque rupture is promoted by:

• accumulation of large amounts of fat and insufficient collagen content in it (a protein that forms the basis of connective tissue);
• inflammation and blood “fluidity” factors.

Risk factors and their control

Angina pectoris and the disease that causes it require not only the fastest possible treatment, but also control of risk factors. These include:

• Hyperlipidemia - imbalance in cholesterol metabolism. It is characterized by an increased concentration of low-density lipoproteins and fatty acids - triglycerides. And also by lowering the level of high-density lipoproteins - “good” cholesterol.

  It is observed in most patients with angina pectoris. Reducing cholesterol levels in the body helps reduce the number of attacks.

• Obesity - characterized by a body mass index of more than 26 and, often, occurs due to constant poor nutrition: consumption of high-calorie foods high in animal fats, cholesterol and “fast” carbohydrates, such as sugar and flour. And, on the contrary, a lack of vegetables, fruits, whole grains, legumes, and herbs in the diet.

  In addition, large body weight increases the load on the heart, increasing the risk of an attack. To control this factor, it is necessary to adhere to the principles of rational or therapeutic nutrition: limit the consumption of cholesterol, animal fats, processed foods and salt. Increase your consumption of vegetables, fruits and other whole plant foods containing fiber, natural antioxidants, polyphenols, and flavonoids.

  Excess body weight is usually just the “tip of the iceberg”. This is a manifestation of serious imbalances in the functioning of the body.

• Physical inactivity - insufficient physical activity, combined with poor nutrition, contributes to the risk of obesity and cholesterol accumulation. Together, these factors contribute to the development and frequency of angina pectoris.
• Arterial hypertension - is a frequent companion to coronary heart disease, which, in itself, implies oxygen deficiency of the myocardium. When blood pressure rises, myocardial tension occurs, causing the heart to have an even greater need for oxygen. This provokes an attack of angina pectoris.

  Controlling blood pressure will help keep the heart and blood vessels relatively calm - reducing the frequency and power of attacks.

• Anemia is not a risk factor, but a condition that aggravates the course and makes it difficult to control IHD. It is characterized by a decreased level of hemoglobin and a general weakening of the body. This results in the impossibility of sufficiently supplying the heart with oxygen. Controlling the normal level of hemoglobin in the blood helps alleviate the course of the disease: for women the norm is 120-150 g/l, for men - 135-165 g/l.
• Tobacco addiction - smoking increases several risk factors at once: it causes oxygen starvation, increases blood pressure and causes spasms of the arteries. The habit harms a healthy body, and in the presence of atherosclerosis it leads to early manifestations of angina pectoris and significantly increases the risk of acute myocardial infarction.
• Diabetes mellitus type 2. This disease seriously worsens the course and prognosis of IHD. We will devote a separate article to this condition, but for now we draw attention to it as a serious risk factor.

The combination of 2 or more items from the list, even weakly expressed ones, increases oxygen starvation of the heart, and, accordingly, the risk of developing and the severity of symptoms of angina pectoris. The presence of risk factors should be taken into account when determining treatment tactics and secondary prevention of angina pectoris.

Coronary heart disease, angina pectoris and rules of life

Usually a well-functioning heart is practically undisturbed, but there may come a time when the heart suddenly makes itself felt.

Most often this is associated with the development of a serious disease – coronary heart disease.

What is coronary heart disease?

IHD - coronary heart disease (angina pectoris, unstable angina, myocardial infarction) - is a consequence of narrowing and blockage of the main arteries of the heart with atherosclerotic plaques. Over time, there are more and more of them, and when the lumen of the vessel is blocked by 50% or more, difficulty in blood flow occurs. As a result, the delivery of oxygen and nutrients to the heart muscle decreases, oxygen starvation (hypoxia) develops, which leads to myocardial ischemia. The larger the size of the atherosclerotic plaque, the smaller the lumen of the vessel and the less blood flows through it, the more pronounced the myocardial hypoxia, which means the more pronounced ischemia (angina).

If, due to physical activity or emotional stress, you experience pain or a feeling of pressure or heaviness behind the sternum, you have a high probability of having angina pectoris (formerly called angina pectoris).

It is strongly recommended that you be examined by a cardiologist or physician.

Know that an attack of angina can be triggered by exercise, emotional stress, cold air and smoking.

Under the influence of these external influences, the pulse (heart rate) increases and blood pressure (BP) increases, which entails an increase in the myocardial oxygen demand against the background of a decrease in its delivery to the heart cells, thus developing myocardial ischemia, which the patient feels as angina pectoris.

How to diagnose angina?

The diagnosis of coronary artery disease and angina is made on the basis of a thorough survey and complaints.

To confirm the diagnosis and clarify the severity of the disease, additional methods are needed:

— taking an electrocardiogram (ECG) at rest and at the height of an attack

— carrying out stress tests (treadle test or test on a bicycle ergometer)

— according to indications (in case of high pulse, abnormal heart rhythm), 24-hour ECG recording is performed (Holter ECG monitoring)

— Sometimes coronary angiography (contrast study of the arteries of the heart) is required.

Warning signs of angina development:

— sudden discomfort, pain, or burning sensation;

— Pain can be triggered by walking, any physical activity, anxiety, emotional stress, cold air, smoking, less often pain appears at rest;

— pain most often occurs behind the sternum, but can radiate (radiate) to the left arm, left half of the lower jaw, teeth, shoulder, back or upper abdomen;

— pain occurs predominantly in the form of mild attacks (3-5 minutes), attacks can be repeated with varying frequency;

— usually the pain disappears 2-3 minutes after stopping walking or other physical activity, or taking nitroglycerin;

— attacks of angina may intensify with increased blood pressure, smoking, late intake or withdrawal of medications.

Types of angina:

V If your attacks appear during the day after the same load, with the same frequency and are of the same type, then you have stable angina.

V If your attacks become more frequent, occur with less stress and even at rest, become stronger, more severe and last longer, and are poorly controlled by the usual dose of nitroglycerin, then you should suspect unstable angina .

You need urgent medical consultation.

V If the pain becomes more intense and lasts for more than 15 minutes, repeats in waves at rest and does not go away after taking three nitroglycerin tablets, there is severe weakness and a feeling of fear, blood pressure and pulse fluctuate sharply, then you should suspect myocardial infarction. In this situation, urgent consultation with a doctor and immediate call for emergency medical assistance is necessary!

To improve your well-being and prevent the development or further progression of IHD, determine whether you have risk factors.

Factors that increase your risk of developing CHD

(check your risk factors):

Factors you cannot control

[] age/gender: men over 45 years old, women over 55 years old

[] early development of IHD in close relatives: - in men under 55 years of age, - in women under 65 years of age

Factors you can control

[] elevated blood pressure

[] smoking

[] elevated blood cholesterol levels

[] elevated blood glucose levels (diabetes)

[] excessive alcohol consumption

[] passive lifestyle

[] overweight

[] stressful situations

Inform your doctor about the risk factors you have identified and ask for advice on what you should do to correct them and subsequently control them well.

Treatment of coronary artery disease and angina has two goals:

the first is to improve the prognosis and prevent the occurrence of serious complications - myocardial infarction, sudden death - and prolong life;

the second is to reduce the severity of clinical symptoms - the frequency and intensity of angina attacks and, thus, improve the quality of life.

You should:

* learn to stop an attack of angina:

— interrupt the load, stop,

— take nitroglycerin under the tongue;

* be informed:

— about the potential undesirable effects of nitrates,

— about the possibility of developing hypotension after taking nitroglycerin;

— about the need to immediately call an ambulance if an angina attack persists at rest and/or is not relieved by nitroglycerin for more than 15-20 minutes;

— about the nature of angina, the mechanism of its development and the significance of this disease in the prognosis of life and recommended methods of treatment and prevention,

* to prevent an attack of angina, take nitrates (nitroglycerin) before exercise, which usually leads to angina.

How can you reduce the high likelihood of progression of coronary artery disease, angina,

as well as the development of serious cardiovascular complications

(myocardial infarction, unstable angina, sudden death, heart failure, cerebral stroke)?

STEP 1 relieving an attack of angina

If you have an angina attack:

V stop physical activity, stop walking, sit down, calm down and relax,

V put one nitroglycerin tablet under the tongue or use nitroglycerin in the form of a spray,

V Before taking nitroglycerin, you should sit down, because the drug may cause dizziness or decreased blood pressure,

V crush the nitroglycerin tablet and let the tablet dissolve completely; if the pain does not stop, then after 3-5 minutes repeat taking nitroglycerin either in the form of a tablet (up to 3 tablets) or in the form of a spray (spraying into the mouth up to 3 times)

V If an attack of angina cannot be relieved within 15 minutes after taking 3 tablets of nitroglycerin, you should immediately seek emergency medical help, and immediately chew 1 tablet of aspirin with water (aspirin prevents the formation of a blood clot). You may be developing a myocardial infarction!

REMEMBER!

You should always have a sufficient number of nitroglycerin tablets with you and they should always be at hand, no matter where you are at the moment!

STEP 2 regular blood pressure checks

People with elevated blood pressure levels and untreated developed myocardial infarction in 68 out of 100 cases and cerebral stroke in 75 out of 100 cases.

Monitor blood pressure levels carefully:

* at least once a year – with a single unstable increase in blood pressure discovered by chance;

* at least once a month - if you feel well, but have a tendency to frequently increase blood pressure;

* at least 2 times a day – in case of exacerbation, poor health, stable increase in blood pressure

Aim for a blood pressure level below 140/90 mmHg.

If you have suffered a myocardial infarction, suffer from angina pectoris, intermittent claudication, then strive for a lower blood pressure level - below 130/80 mm Hg.

In people with good blood pressure control:

* 42% fewer brain strokes;

* 20% less lethal and other serious cardiovascular complications (myocardial infarction, unstable angina, sudden death);

* 50% less heart failure;

* 14% fewer deaths from any other non-cardiac cause.

STEP 3 regular monitoring of cholesterol levels in the blood

Check your blood cholesterol levels and, if they are elevated, discuss with your doctor the possibility of taking cholesterol-lowering medications.

Criteria for optimal lipid levels in the majority of patients with coronary artery disease, patients who have had myocardial infarction, or surgery on the vessels of the heart, carotid arteries or arteries of the lower extremities:

* Total cholesterol - less than 4.0 mmol/l

* LDL cholesterol – less than 1.8 mmol/l

* Triglycerides – less than 1.7 mmol/l

* HDL cholesterol – for Men – 1.0 mmol/l or more

* HDL cholesterol - for Women - 1.2 mmol/l or more

STEP 4 heart rate control

In case of coronary artery disease, angina pectoris, or after a myocardial infarction, it is very important to control the pulse rate (heartbeat).

The optimal heart rate should be between 55-60 beats per minute.

This optimal heart rate is usually maintained with special medications prescribed by the attending physician.

STEP 5 proper nutrition

Change your eating pattern: eat healthy foods with less animal fat (60-105 g/day for men and 45-75 g/day for women) and rich in vegetables, fruits, complex carbohydrates, dietary fiber, fish.

Exclude from the diet: fatty meats, strong meat broths, any fat, sausages (even the so-called doctor's ones!), sausages, ready-made dumplings, canned meat, mayonnaise, internal organs of animals, brains, caviar, butter, hard margarine, fatty cottage cheese , cream cakes, creamy ice cream.

STEP 6 more movements

Move more and be physically active (at least 30 minutes of moderate exercise on most days of the week). If you have heart problems and are taking medications, talk to your doctor before changing your level of physical activity.

If you have been physically inactive for a long time, or if you are over 40 years old, or have angina pectoris, then the mode and level of physical activity should be determined by your doctor or physical therapy specialist, determining the pulse safety zone.

Physical activity is useful: aerobic, dynamic and moderate intensity - cycling, measured walking, swimming, team sports (football, volleyball), etc.

Optimal training walking tempo (number of steps per minute) depending on physical load tolerance (according to a load test on a bicycle ergometer)

Amount of load performed

during bicycle ergometer test, W 50 75 100 125

Function class

angina III II II I

Optimal pace

number of steps per minute 99-102 105-108 111-114 118-121

(or average) or 100 or 105 or 110 or 120

STEP 7 monitor the status of other risk factors

> Monitor your weight and measure your waist circumference (a waist circumference greater than 102 cm for men and greater than 88 cm for women indicates abdominal obesity, which is associated with a risk of cardiovascular complications).

> Stop smoking.

> Learn to control your psycho-emotional stress (avoid conflict situations, devote time to auto-training and relaxation methods).

> Maintain normal blood glucose levels: < 6 mmol/L (110 mg/dL)

STEP 8 Take medications prescribed by your doctor regularly

Today, the doctor has at his disposal a sufficient number of highly effective and safe drugs to reduce high blood pressure, prevent angina pectoris and prevent the development of serious cardiovascular complications.

REMEMBER!

Interruptions in taking medications reduce the effectiveness of preventing the development of myocardial infarction, unstable angina, and heart failure.

For the treatment of coronary artery disease and angina pectoris, different classes of medications are used today, the action of which is aimed at preventing the development of myocardial ischemia and angina attacks, vascular thrombosis, preventing the progression of atherosclerosis, reducing blood pressure and pulse rate (heart rate).

Take medication as directed by your doctor!

If the disease, despite active drug therapy, progresses, it is necessary to use modern interventional and surgical methods to restore blood supply to the myocardium:

V balloon angioplasty – expansion of the vessel at the site of stenosis by inflating the balloon

V stenting of the coronary arteries - installation of a special stent (tube) that allows for a long time to expand the artery of the heart and ensure normal blood flow

V coronary artery bypass grafting is used for a certain localization of atheromatous plaque and significant and multiple narrowing of the coronary vessel, when the first two methods are not effective

MAIN!

Treatment of coronary heart disease through lifestyle changes and highly effective medications prescribed by a doctor must be constant, long-term and daily.

STEP 9 keep a self-monitoring diary

A self-monitoring diary must be kept in order to promptly notice negative changes in the course of the disease and immediately contact your doctor.

The diary should record: blood pressure level, pulse rate, number of angina attacks, amount of nitroglycerin in the form of tablets or spray inhalations that were taken to relieve (relieve) an angina attack or prophylactically, for example, before physical activity, going outside to prevent it development.

The diary will allow you and your attending physician to evaluate the effectiveness of treatment, prevent exacerbation of the disease and, if necessary, correct therapy.

IF YOU FOLLOW THESE RULES AND MANAGE YOUR LIFE SMARTLY, YOUR ILLNESS WILL ALWAYS BE UNDER GOOD CONTROL!

Diagnosis of angina pectoris

To diagnose angina pectoris, as well as exclude other possible diseases, complaints and the nature of pain are analyzed, laboratory blood tests and a functional examination of the heart are performed, and the patient’s physical condition is analyzed.

It is also important to determine the severity and severity of the process. To do this, if a diagnosis of acute coronary syndrome is made, a study of the vascular bed of the heart is performed - coronary angiography. Based on the results, the question of surgery or intravascular intervention is decided.

Laboratory blood diagnostics help determine the level of:

• total cholesterol and lipid fractions,
• high and low density lipoproteins,
• triglycerides,
• creatine kinase,
• glucose,
• markers of systemic inflammation (SII),
• indicators of the coagulation system - coagulograms,
• electrolytes,
• troponin - in acute situations.

Particular attention in the study is paid to the presence of cardiac troponins I and T - markers that indicate myocardial damage. Their presence confirms that the patient is suffering a microinfarction or acute myocardial infarction.

Functional research methods allow you to determine the condition of the heart:

• ECG - examination at the peak of an angina attack determines conduction and rhythm disturbances. Signs of myocardial oxygen starvation may also be detected.
• Daily ECG monitoring - records the level of oxygen starvation during an attack of angina, heart rate, arrhythmia. Indicators help determine the specific diagnosis and stage of the disease.
• EchoCG - reveals local ischemic changes and disorders of myocardial contractility.
• Bicycle ergometry or treadmill - determines the maximum possible physical activity for the patient without the threat of developing ischemia. The load is set using an exercise bike or treadmill, and the results are recorded by an ECG device.
• Stress echocardiography (Stress-ECHO) is a study of myocardial contractility using the ultrasound method, under conditions of dosed physical activity. At the same time, an ECG is recorded. Allows you to make a primary diagnosis of IHD and clarify indications for surgical treatment.

For a detailed study of the vascular bed of the heart, the following are used:

• Coronary angiography is an X-ray method in which a contrast agent is injected into the arteries of the heart using a special catheter.
• multislice computed tomography is a type of computed tomography in which X-ray “slices” of the heart and blood vessels are obtained at high speed. More than 100 such “slices” can be performed between contractions of the heart chambers.

These studies allow us to obtain information about the state of the vascular bed of the heart: which arteries are passable, which are completely or partially blocked, and in what place. Based on the information received, the doctor makes a decision about whether the patient has coronary artery disease, the need for surgical intervention and the timing of treatment.

Treatment of angina

Angina pectoris is an accompanying factor of coronary heart disease, so the main fight must be waged against it. Treatment of angina pectoris is aimed at relieving and preventing attacks and complications.

The first priority when an attack occurs is to take nitroglycerin. It helps relieve pain. If the attack does not stop, nitroglycerin can be used again. Drug therapy involves your doctor prescribing anti-ischemic drugs that help the heart cope with low oxygen levels. Anti-sclerotic drugs, antioxidants and antiplatelet agents are also prescribed.

Based on the diagnosis, a decision is made on additional treatment for cardiac conduction and rhythm disorders. For angina pectoris, surgical intervention is often prescribed: balloon angioplasty, stenting, coronary artery bypass grafting.